The drugs are given over the counter, but can still take quite a while. Diet and exercise are very important for treating Huntington's as they prevent many of the symptoms of the disease. Exercise has been shown to improve the functioning of the brain and also reduces the amount of glutamate made, and can help people to recover faster from the disease. It is also said that the more Parkinson's patients there are, the more effective the drug regimen will be to keep them alive. Diaminodiphenyl sulfone the Disease of Aging Huntington's will eventually cause the body to stop making new neurons. It does this by blocking the dopamine and norepinephrine neurotransmitter system.
The brain and the rest of the body eventually die, and people who are dying of the disease can often feel this. Gluconozyme breaks down all of the glutamine to form toxic substances that cause the neurons to die. The diaminodiphenyl sulfone itself is not toxic, but it causes a condition known as degeneration, which is characterized by the loss of the neurons' normal ability to maintain proper function. These cells die because the damage done to the neurons by the protein prevents it from doing its job properly. A study published earlier this year identified a new genetic disease in which the brain's nerve cells begin to die from the damage the disease causes. The study was described in a paper published this week in the journal Science. There are some caveats to the study, however. It is not clear how long the mice stay disease-free after being infected with the mutant protein.
It is also very early in the process to start looking at the potential effects of the mutations on humans, and how they might cause harm, which is where this study will need to be carried out. The team used mice infected with the mutation. It is possible this will be useful to researchers looking at diseases involving the brain, for example, Alzheimer's. But the mouse study was designed to explore the issue, not to help humans get rid of the genetic disease. The researchers used a genetic modification to turn on the gene, and this has been shown in mice to lead to some damage to the nerve cells.
It should be noted that the study only looked at what happened to the damaged nerve cells-- it does not determine whether these damage are permanent. The mutation is found in a group of genes called SLC6A3, also known as the SLC6A24 gene. It is the same gene associated with the SLC6A11 gene for alcohol and the K562 gene for nicotine, which is responsible for the addictive properties of some tobacco products. The new gene also causes some damage in the nerve cells.